Effect of chronic moxonidine therapy on peripheral sympathetic activity in essential hypertension
نویسندگان
چکیده
منابع مشابه
Chronic I(1)-imidazoline agonism : sympathetic mechanisms in hypertension.
Evidence exists for a state of sympathetic hyperactivity in essential hypertension, and moxonidine, a new central sympathetic inhibitor, has been introduced for its treatment. Acute administration of moxonidine lowers peripheral sympathetic neural output. This study examined the effect of chronic moxonidine therapy, at increasing therapeutic doses, on resting peripheral sympathetic activity and...
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Neural Sympathetic Activity in Essential Hypertension To the Editor: We read with great interest the article by Schlaich et al1 dealing with the issue of sympathetic augmentation in hypertension. In 1993, we demonstrated that neural sympathetic activity predominance is registered in essential hypertensive patients.2 These findings were supported by the overwhelming circulating noradrenaline (NA...
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Understanding the connection between sympathetic activity and essential hypertension is still rudimentary. We studied interrelationships of plasma catecholamines, plasma renin activity (PRA), aldosterone, sodium intake, and therapeutic response of 20 normal renin hypertensives. Based on plasma norepinephrine (NE), this population fell into two distinct subsets. The 11 patients in the "normal" N...
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15 صفحه اولArterial pressure lowering effect of chronic atenolol therapy in hypertension and vasoconstrictor sympathetic drive.
Although the beta1-adrenergic blocking agent atenolol is an established antihypertensive therapy, its effect on peripheral sympathetic vasoconstrictor drive has remained controversial. In patients with hypertension, atenolol therapy has been reported to either increase or have no effect on peripheral vascular resistance, despite other reports showing no change or a decrease in peripheral sympat...
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ژورنال
عنوان ژورنال: American Journal of Hypertension
سال: 1999
ISSN: 0895-7061
DOI: 10.1016/s0895-7061(99)80091-0